Subclavian Steal in Patients with Blalock-taussig Anastomosis.
نویسندگان
چکیده
ALTHOUGH the collateral anastomotic channels that occur secondary to obstruction of the first segment of the subclavian artery have received relatively little comment by anatomists, the participation by the vertebral artery in such instances has been mentioned.' Recently, the finding of retrograde flow from the vertebral artery on the side of an obstruction in the first part of the subclavian artery to the distal segment of that subelavian artery has been noted by several investigators.2-10 Anatomically, obstruction may be complete or, if partial, of sufficient severity to lower the pressure in the subclavian artery to below that in the vertebral artery. The reversal of flow, demonstrable angiographically, may cause basilar artery insufficiency giving rise to symptoms of headache, visual disturbance, paresthesia, hemiparesis, dizziness, and syncope. This condition, first reported by Reivich and co-workers,2 has been termed the "subelavian steal syndrome."" With the exception of one report,12 the subelavian obstruction in all the cases of the entity recorded to date has been on the basis of atherosclerotic disease within the vessel. Anastomosis of subelavian artery to pulmonary artery13 has been performed since 1945 on a large number of patients suffering from cardiac anomalies associated with pulmonary stenosis or atresia. On the assumption that patients treated in this manner might exhibit the subelavian steal angiograms
منابع مشابه
Short Communications Vertebrobasilar Ischemia After Total Repair of Tetralogy of Fallot: Significance of Subclavian Steal Created by Blalock-Taussig Anastomosis Vertebrobasilar Ischemia After Correction of Tetralogy of Fallot
Patients who have undergone a Blalock-Taussig anastomosis for treatment of congenital heart disease may have the vascular anatomy of the subclavian steal syndrome. Cerebral ischemia has been reported in such patients, but not when total surgical correction has eliminated other predisposing factors. We report a patient who developed vertebrobasilar insufficiency 31 years after Blalock-Taussig an...
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ورودعنوان ژورنال:
- Circulation
دوره 31 شماره
صفحات -
تاریخ انتشار 1965